MedicED EMS Continuing Education approved by CAPCE
This is a printable version for reference only.
Please view full version on www.MedicEd.com for images and videos associated with this course
MedicEd.com - xHyperbarics - NEED TO REVISE
Lecture



Hyperbarics
 

By: Michelle E. Duffelmeyer, MD  and Ellen Smithline, RN


Overview
This lecture will review the basic principles that govern the effects of air pressure on gases. Potential problems can occur at every phase of ping. Diving injuries that occur on the surface, during descent, on the bottom, and during ascent will all be reviewed. The mechanism of action, evidence for and risks of treatment with hyperbaric oxygen therapy will also be reviewed in this lecture. 

 


Course Approved for the Following Levels:
First Responder
EMT Basic
EMT Intermediate
EMT Paramedic 



Objectives
 
By the end of this lecture, the participant should be able to...
  1. Describe the basic principles that explain the effects of air pressure on gases – Boyle’s Law, Dalton’s Law, Henry’s Law
  2. Describe the spectrum of ping injuries including injuries on water’s surface, injuries during descent, injuries on the bottom and during ascent
  3. Explain the management of ping emergencies in general
  4. Describe specific treatment plans for the most serious ping emergencies – decompression illness, pulmonary over-pressure accidents, arterial gas embolism, pneumomediastinum, pneumothorax and nitrogen narcosis
  5. Explain the basics of hyperbaric oxygen therapy
  6. Be familiar with the Divers Alert Network


Scuba
 
  • SCUBA ping is a popular recreational sport.
  • More than 250,000 new pers are certified annually.
  • SCUBA is an abbreviation for Self-Contained Underwater Breathing Apparatus.
  • With more and more inexperienced pers, the risk increases for ping emergencies.
 
scba.JPG

References

 

1. Kizer, K. Diving medicine. Emergency Med Clin North Am. 1984; 2:513-530.



Mechanical Effects of Pressure on Gases
 
  • To understand how pe injuries occur is it essential to understand 3 physical laws that explain the effects of pressure on gases.


Boyle’s Law
 
  • The volume of gas in an enclosed space is inversely proportional to the pressure exerted on it.
  • In effect, as pressure increases, volume decreases. An example would be a balloon.
  • If a balloon were inside a chamber, as pressure increases the volume of the balloon decreases. Deflating the balloon. As the pressure inside the chamber decreases, the balloon size would expand, and the volume would increase.

 

 

 

 

References

1. Tibbles, P., & Edelsberg, J. S. (1996). Hyperbaric-Oxygen Therapy. New Eng J Med, 334, (25).



How Boyle’s Law Applies to Seawater
 

In thinking about SCUBA ping emergencies, realize that saltwater has a density and this density can be equated to pressure. As a per goes deeper and deeper the pressure increases. As Boyle’s Law states, the volume of air is therefore compressed. At 66 feet underwater, one liter of air would be compressed to 333 1/3rd mL.

 

 

 

References

  1. Richardson, D. (1999). Open Water Diver Manual PADI. Santa Margarita, CA: International PADI, Inc.


Dalton's Law
 
  • The total pressure of a mixture of gases is equal to the sum of the partial pressure of the inpidual gases.
  • At greater depths under the sea the total pressure will increase but each inpidual gas will still account for the same proportion of that total pressure. For example, nitrogen 78%, oxygen 21%, and carbon dioxide less than 1%.

 

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.


Henry’s Law
 
  • Henry’s Law states that the amount of gas dissolved in a given volume of fluid is proportional to the pressure of the gas above it.
  • This seems like high school physics all over again but this law has important implications for SCUBA pers.
  • Lets review what happens to nitrogen and oxygen as a per descends.
  • Both gases tend to dissolve in liquid (blood plasma) with the increased pressure of the water bearing down. Oxygen is used by the tissues in metabolism so little is left dissolved in the blood and tissues. Nitrogen is an inert gas and is not used by the body. Nitrogen dissolves.

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.


What Happens to the Nitrogen?
 
  • As the per descends, the nitrogen is dissolving in the blood and the tissues of the per’s body. When the per now ascends toward the surface (sea level) again the gases that were dissolved are now under less pressure and come out of the blood and tissues.
  • Nitrogen is the main gas involved. If the ascent is too rapid, the nitrogen will come out of the blood and tissues as bubbles. You can think of it as unscrewing the top of a soda bottle too quickly. The gas rushes to form bubbles and rise out of the bottle.


Pathophysiology of Dive Emergencies
 
Now that you can understand the basic laws of physics and how they apply to gases under pressure, we can review what happens when a per gets in trouble. Accidents can occur at all 4 stages of ping:
  1. On the surface
  2. During descent
  3. On the bottom
  4. During ascent

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.


Surface Injuries
 
  1. Boating accidents
  2. Entanglement of lines or in kelp beds
  3. Fatigue
  4. Cold injury producing shivering or blackout
  5. Marine life – sharks, eels, man-o-war


Injuries During Descent
 
  • Barotrauma injuries occur at this phase of ping – “the squeeze.”
  • Barotrauma is injury due to the pressure. To equilibrate pressure between the middle ear and the nasopharynx, a per needs a patent eustachian tube. Do not pe if you have an upper respiratory infection.
  • Problems at this stage of ping can be:
  1. Middle ear pain
  2. Tinnitus – ringing in the ear
  3. Dizziness
  4. Hearing loss
  5. Tympanic membrane (eardrum) rupture
  6. Severe headaches around the frontal or maxillary sinuses

 

 

References

  1. Richardson, D. (1999). Open Water Diver Manual PADI. Santa Margarita, CA: International PADI, Inc.


Injuries at the Bottom
 

While at the bottom, pers can be caught in the “raptures of the deep.” This is really a description for nitrogen narcosis

  •  narcosis is a state of stupor that develops secondary to nitrogen’s anesthetic effect on cerebral function.

  •  A per may take dangerous risks feeling like he or she intoxicated.

 

Other injuries at the bottom may be…

1. Running out of air

2. Marine life injury

 

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Richardson, D. (1999). Open Water Diver Manual PADI. Santa Margarita, CA: International PADI, Inc.


Injuries During Ascent
 

  1. Barotrauma can occur again during ascent as pressures are changing.
  2. Decompression illness or “the bends.” Divers going below 40 feet are required to do a staged ascent to prevent the nitrogen from forming bubbles. If the ascent is too rapid, nitrogen will form bubbles causing severe pain especially in the abdomen and in the joints. This is the bends.
  3. Pulmonary over-pressure. Air compressed in the lungs expands and can rupture alveoli if not exhaled. This can happen if the per holds his or her breath during ascent.
  4. Arterial gas embolism. An air bubble that enters the blood stream from a damaged lung.
  5. Pneumomediastinum. Air (gas) that ruptures through the pleura of the lungs into the center of the chest putting pressure on major vessels and the heart.
  6. Pneumothorax. Rupture of alveoli causing air (gas) to be in the space surrounding the lung placing pressure and collapsing the lung.

 

 

References
  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Rosen, P. et al. (1992). Emergency medicine concepts and clinical practice. St. Louis, MO: Mosby-Year Book Inc. pp. 985-993.


Management of Diving Emergencies
 

  1. History is critical. At what phase of ping did the injury occur?
  2. Timing of symptoms. When were symptoms first noted? This can help pinpoint whether the injury is related to ascent, descent, or at the bottom.
  3. Type of breathing equipment used.
  4. Garments warn. Is hypothermia an issue?
  5. Parameters of the pe – The 3 D’s - depth, pes, duration.
  6. What was the depth of the pe, the number of pes, and the duration of the pe/s?
  7. Rate of ascent.
  8. Was panic a factor? Did that cause the ascent to be too rapid?
  9. Diver’s experience
  10. Old injuries
  11. Medications
  12. Alcohol

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Rosen, P. et al. (1992). Emergency medicine concepts and clinical practice. St. Louis, MO: Mosby-Year Book Inc. pp. 985-993.



Decompression Illness “The Bends”
 
Nitrogen bubbles collecting in the joints, tendons, inner ear, spinal cord, skin and brain cause decompression illness. It occurs 15 minutes to 12 hours after a pe. Decompression illness can come on gradually and persist or symptoms can be intermittent.


Signs and Symptoms of Decompression Illness
 
  • Abdominal pain
  • Joint pain
  • Fatigue
  • Paresthesias
  • Paralysis
  • Shock
  • Weakness
  • Breathing difficulties

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Richardson, D. (1999). Open Water Diver Manual PADI. Santa Margarita, CA: International PADI, Inc.


Treatment of Decompression Illness
 

  • Assess ABC’s
  • CPR if needed.
  • 100% O2 by non-rebreather if conscious; otherwise intubate.
  • Place patient in supine position.
  • IV fluids – NS or Lactated Ringer’s. IV fluid should be in collapsible plastic bags. No glass. (glass can break in the chamber)
  • Any catheters should be filled with saline not air.
  • Rapid transport to nearest ER or facility with hyperbaric oxygen chamber. The longer recompression therapy is delayed the higher the morbidity.
  • If transported by un-pressurized helicopter, the pilot should fly as low as possible or cabin pressure should be maintained at sea level.
  • Contact the Diver’s Alert Network if unsure of the nearest location of a recompression chamber.

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Rosen, P. et al. (1992). Emergency medicine concepts and clinical practice. St. Louis, MO: Mosby-Year Book Inc. pp. 985-993.



Arterial Gas Embolism (AGE)
 

Arterial Gas Embolism (AGE) is caused by pressure building up in the lungs and subsequent alveoli rupture allowing an air bubble to enter the circulation. Once in the circulation, the air embolism can obstruct blood flow leading to ischemia, infarct and stroke. The onset can occur within 2 to 10 minutes of ascent.

 

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.



Signs and Symptoms of Arterial Gas Embolism
 

  • Sharp tearing pain
  • Focal neurological deficits
  • Confusion
  • Vertigo
  • Loss of consciousness
  • Visual disturbances

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Rosen, P. et al. (1992). Emergency medicine concepts and clinical practice. St. Louis, MO: Mosby-Year Book Inc. pp. 985-993.



Treatment of AGE
 

 

  • Treat AGE as with decompression illness.
  • Place patient in lateral decubitus/Trendelenburg position to minimize the chance the air embolism will go to the heart or brain.
  • Again, rapid transport to recompression chamber by pressurized aircraft or low altitude flying.

 

 

References

1.  Rosen, P. et al. (1992). Emergency medicine concepts and clinical practice. St. Louis, MO: Mosby-Year Book Inc. pp. 985-993.



Treatment of Pneumomediastinum/pneumothorax
 
  • 100% O2 via non-rebreather
  • IV lactate Ringer’s or normal saline
  • Rapid transport


Signs and Symptoms of Nitrogen Narcosis
 
Altered levels of consciousness and impaired judgment are the classic symptoms.


Treatment
 

  • Simply return to shallow depth. This resolves on ascent. To avoid this problem in deep pes a mixture of oxygen and helium is used. Helium does not cause the anesthetic effect of nitrogen.
  • Since treatment of many pe related injuries includes hyperbaric oxygen therapy (HBO) lets review the basics of this treatment.

  • HBO is indicated for other reasons besides SCUBA emergencies and decompression illness.

  • The remainder of this lecture will briefly review the fundamentals of HBO and its clinical applications.

 

 

References

  1. Bledsoe, B. E., Porter, R. S., & Cherry, R. A. (2001). Paramedic Care: Principles & Practice. Upper Saddle River, NJ: Prentice-Hall, Inc. pp 528-536.
  2. Richardson, D. (1999). Open Water Diver Manual PADI. Santa Margarita, CA: International PADI, Inc.



Hyperbaric Oxygen Therapy
 
 Hyperbaric Oxygen Therapy is defined as
  • Breathing 100% oxygen intermittently while in a pressurized environment.
  • It can be compared to scuba ping without the water.
A patient can breath 100% oxygen with a
  • Mask
  • Hood
  • Endotracheal tube
  • Or ambient air depending on the type of hyperbaric chamber used.


Hyperbaric Oxygen Chambers
 
Your facility can be equipped with either a monoplace or multiplace hyperbaric chamber.


Monoplace chamber
 
 A monoplace chamber
  • Is pressurized with 100% oxygen, not air.
  • Size allows only one adult or possibly 2 children to occupy the chamber.
  • Advantage is that it does not require a health care attendant (i.e.; physician, nurse, RRT, or EMT) to accompany the patient.
  • Disadvantage is the there is no “hands on” contact.


Multiplace Chamber
 
 The multiplace chamber
  • Pressurized with air.
  • Patient breathes 100% oxygen via hood, mask, or endotracheal tube.
  • Two or more patients, depending on the size of the chamber, plus a health care attendant can be treated at one time.
  • Advantage - allows for a hands on ICU environment.
  • Disadvantage -A full complement of staff is required.
  • The attendants are exposed to a pressurized environment while accompanying the patient(s), therefore exposing them to a risk of decompression sickness.
  • The number of monoplace chambers exceeds the number of multiplace chambers in the United States.


Mechanism of Action
 
 Hyper-oxygenation:
  • Remember, the patient breathes 100% oxygen while under pressure, therefore increasing the oxygen content of blood by increasing tissue oxygen tensions via the plasma and hemoglobin.
  • Help produce small blood vessels known as capillaries.
  • It can also act as an antibiotic against certain organisms that thrive on low oxygen levels.
  • Thought to increases cell production quicker.


Mechanism of Action Continued
 
  • Normal arterial oxygen (paO2) levels in the body are 80mmHg-100mmHg. HBO can allow the body to increase its oxygen uptake to a maximum of 2183mmHg during a treatment.
  • This effect is used for indications such as carbon monoxide poisoning, cyanide poisoning, burns, severe anemia, gas gangrene, crush Injury, compartment syndrome, other acute traumatic Ischemia and adjunctive therapy for enhancement of healing in selected problem wounds.


Hyperbaric Oxygen (HBO) Therapy is an ADJUNCTIVE treatment.
 
 Continue conventional therapy
  • BCLS
  • IV
  • Debridement if indicated
  • Wound care if indicated


 
  • Air or Gas Embolism
  • Carbon Monoxide Poisoning and Smoke Inhalation
  • Cyanide Poisoning
  • Clostridial Myonecrosis (Gas Gangrene)
  • Crush Injury, Compartment Syndrome and other Acute Traumatic Ischemia
  • Decompression Sickness


Air /Gas Embolism
 
 Acquired iatrogenically (in hospital) by
  • Central line insertion
  • O.R. procedure such as coronary arterial bypass graft
  • Other procedures


Also can occur with
  • Collapsed lung
  • Scuba ping
  • Swimming
  • Oral Sex


Effects of HBO on Air /Gas Embolism
 
  • Decreases size of air/gas embolism as pressure increases in chamber
  • Hyper oxygenate hypoxic tissues


Carbon Monoxide (CO) Poisoning
 
  • Product of incomplete combustion
  • Can be produced by burning any fuel
    • Gasoline
    • Propane
    • Natural gas
    • Oil
    • Wood
    • Coal
  • CO (carboxyhemoglobin) levels build up in blood in relation to amount in air and time of exposure
  • A group of people in same area of exposure for same length of time can have varying levels and different presentation of symptoms
  • fire.JPG


    Effects of HBO on Carbon Monoxide Levels
     
    • Decrease the half life of carboxyhemoglobin levels. This means faster clearing of carbon monoxide.
    • Hyper-oxygenates hypoxic tissues


    Clostridial Myonecrosis (Gas Gangrene)
     
    • A toxin that thrives in arterial levels of 300 mmHg or less
    • Spreads 1 – 6 inches an hour
    • Effects of HBO on Clostridial Myonecrosis
      • Hyper-oxygenate hypoxic tissues


    Crush Injury, Compartment Syndrome and other Acute Traumatic Ischemia
     
    • Acute insult to tissues leading to hypoxia caused by trauma and/or circumferential constriction of tissues

    • Effects of HBO on Crush Injury, Compartment Syndrome and other Acute Traumatic Ischemia
      • Hyper-oxygenate hypoxic tissues


    Decompression Sickness
     
    • Nitrogen bubble formation that can develop in areas such as blood stream, spinal fluid, joint areas, inner ear and others
    • Found in SCUBA pers, attendants in the hyperbaric environment or High Altitude Flying
    • Can occur even after the per is out of the water, especially if flying within 12 hours of the pe
    • Effects of HBO on Decompression Sickness
      • Decreases size of air/gas embolism as pressure increases in chamber
      • Hyper-oxygenate hypoxic tissues


    Transportation of Patient
     
    • Can be transported by car, ambulance, helicopter, or plane UNLESS PATIENT IS DIAGNOSED WITH DECOMPRESSION SICKNESS OR AIR/GAS EMBOLISM!
    • Increase in altitude will decrease barometric pressure therefore increasing bubble size and increasing symptoms
    • Can only be transported in surface transportation, low altitude helicopter or in a pressurized aircraft which does not exceed a cabin atmosphere of 800 feet of altitude. Commerical airline flights are not rated for transport since they exceed this cabin atmosphere.


    Hyperbaric Medicine Resources
     
    Divers Alert Network (DAN)
    • A nonprofit organization associated with Duke University Medical Center that has ability to refer SCUBA pers and other people in need of a hyperbaric chamber facility.
    • www.persalertnetwork.org
    • Dan’s Diving Emergency Hotline can be reached 24 hours a day, 365 days a year at (919) 684-4DAN or you can call collect at (919) 684-8111.


    Hyperbaric Medicine Resources Continued
     
    Undersea and Hyperbaric Medical Society (UHMS)
    • Professional organization including physicians, nurses, respiratory therapist, pers and other groups
    • Reviews scientific literature
    • Recommends guidelines for use of HBOT
    • www.UHMS.org


    Hyperbaric Medicine Resources Continued
     
    Baromedical Nurses Association (BNA)
    • Professional organization which includes nurses.
    • Reviews scientific literature.
    • Recommends nursing guidelines for use of HBOT.
    • www.hyperbaricnurses.com


    Conclusion
     

    In conclusion, hyperbaric medicine is being utilized to help both improve and save lives of patients diagnosed with these diseases/conditions. As research continues in this field, advances will be made to understand more fully the potential of hyperbaric oxygen therapy.

     

     

     

    References

    1. Davis, JC and TK Hunt. Hyperbaric Oxygen Therapy. Undersea Medical Society, Bethesda, 1977.
    2. Kindwall, E. Hyperbaric Medicine Practice. Best Publishing Company, Flagstaff, 1994.
    3. Shilling, C and C Carlston, et all. The Physician’s Guide to Diving Medicine. Undersea Hyperbaric Medical Society, Bethesda, 1984.



    Additional References
     
    1. Davis, J. C. & Hunt, T. K. (1977). Hyperbaric Oxygen Therapy. Undersea Medical Society, Bethesda, MD.
    2. Kindwall, E. (1994). Hyperbaric Medicine Practice Best Publishing Company, Flagstaff.
    3. Shilling, C. & Carlston, C. (1984). The Physician's Guide to Diving Medicine . Undersea Hyperbaric Medical Society, Bethesda, MD.
    4. Rosen, P. (1992). Emergency Medicine Concepts and Clinical Practice. Mosby Year Book, St. Louis, MO.
    5. Richardson, D. (1999). Open Water Dive Manual. International PADI, Inc., Santa Margarita, CA.
    6. Tibbles, P. & Edelsberg, J. S. (1996). Hyperbaric-Oxygen Therapy. New England Journal of Medicine, 334,25.
    7. An Overview of Hyperbaric Medicine. Patient Care, July 15, 2000.
    8. Ernst, A. & Zibrak, J. D. Carbon Monoxide Poisoning. New England Journal of Medicine, 339, 22.
    9. Bledsoe, B. E., Porter, R. S. & Cherry, R. A. (2001). Paramedic Care: Priniples and Practice Medical Emergencies. Prentice-Hall Inc., Upper Saddle River, NJ.
    10. Good Pressure: What you need to know about hyperbaric chambers. (November/December 1999). Alert Diver: The Magazine of Divers Alert Network.
    This is a printable version for reference only.
    Please view full version on www.MedicEd.com for images and videos associated with this course